Young ovine death during hyperimmunization: crotalic envenomation or copper toxicosis?
نویسندگان
چکیده
منابع مشابه
Sodium fluoride/copper naphthenate toxicosis in cattle.
Fourteen cattle on a Kansas pasture died from ingestion of a wood preservative compound containing sodium fluoride and copper naphthenate. Clinical signs included depression, anorexia, ataxia, diarrhea, and recumbency. Grossly visible lesions included perirenal edema, pale kidneys, and forestomach ulceration. All 3 cows that had postmortem evaluations had extensive renal cortical tubular necros...
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Sheep were sensitized by repeated infection with Haemonchus contortus L3, followed by a 12 week rest period, and an abomasal cannula was surgically implanted in all sheep. Seven of the sensitized sheep were subsequently challenged with 50 000 H. contortus L3 while 4 control sheep were challenged with saline. Biopsy samples were taken using a fibreoptic endoscope on days 0, 1, 2, 3, 5, 7 and 28 ...
متن کاملNew canine models of copper toxicosis: diagnosis, treatment, and genetics.
The One Health principle recognizes that human health, animal health, and environmental health are inextricably linked. An excellent example is the study of naturally occurring copper toxicosis in dogs to help understand human disorders of copper metabolism. Besides the Bedlington terrier, where copper toxicosis is caused by a mutation in the COMMD1 gene, more complex hereditary forms of copper...
متن کاملUse of 64copper measurements to diagnose canine copper toxicosis.
Inherited canine copper toxicosis is a serious problem in Bedlington terriers and West Highland White terriers, and may also be a problem in other less-studied breeds. Affected dogs become ill at midlife with progressive and ultimately fatal liver disease. Treatments for removal of copper and prevention of copper accumulation are available, but are most effective if begun before the dog becomes...
متن کاملDNA testing for copper toxicosis in Bedlington terriers.
Copper toxicosis is an hereditary disease in which failure of the liver to expel dietary copper leads to a build-up of this toxic metal causing illness and death. It is inherited as an autosomal recessive trait. As such, two copies of the defective gene, one inherited from each parent, need to be present before an individual displays clinical signs. Dogs with one copy of the defective gene and ...
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ژورنال
عنوان ژورنال: Journal of Venomous Animals and Toxins including Tropical Diseases
سال: 2008
ISSN: 1678-9199
DOI: 10.1590/s1678-91992008000400015